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Glucotoxicity results in apoptosis in H9c2 cells via alteration in redox homeostasis linked mitochondrial dynamics and polyol pathway and possible reversal with cinnamic acid
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Statistical analysis All experiments were performed independently at least three times and the data were shown as mean and S.E.M
OS primarily affects three key mechanisms: (1) up-regulation of Na/K-ATPase, (2) increases water permeability while simultaneously reducing the protein reflection coefficient through the peritoneal barrier, and (3) stimulation of angiotensin-induced aldosterone secretion (137)
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